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Diethylcarbamazine attenuates LPS-induced acute lung injury in mice by apoptosis of inflammatory cells
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文摘
Diethylcarbamazine (DEC) pretreatment reduced lung tissue damage. DEC inhibited the production of inflammatory markers in the lungs and BALF. DEC prevents NF-κB activation and IkBa phosphorylation. DEC attenuates neutrophil accumulation by stimulation of apoptosis. DEC accelerates the resolution of inflammation and may be a potential pharmacological treatment for ALI.

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