Early life low intensity stress experience modifies acute stress effects on juvenile brain cell proliferation of European sea bass (D. Labrax)
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- 作者：S. Fokosa ; M. Pavlidisb ; T. Yiotisa ; A. Tsalafoutab ; c ; N. Papandroulakisc ; C.R. Dermona ; dermon@upatras.gr" class="auth_mail" title="E-mail the corresponding author
- 关键词：ANOVA ; analysis of variance ; AS ; acute stress ; BLBP ; brain lipid binding protein ; BrdU ; bromodeoxyuridine ; DAB ; 3.3-diaminobenzidine ; Dl ; lateral zone of the dorsal telencephalic area ; Dm ; medial zone of dorsal telencephalic area ; dph ; days post-hatch ; GABA ; gamma-aminobutyric acid ; GR ; glucocorticoid receptor ; gr-1 ; glucocorticoid receptor 1 gene ; gr-2 ; glucocorticoid receptor 2 gene ; mr ; mineralocorticoid receptor gene ; HPA ; hypothalamus-pituitary-adrenal ; HPI ; hypothalamus-pituitary-interrena
- 刊名：Behavioural Brain Research
- 出版年：2017
- 出版时间：15 January 2017
- 年：2017
- 卷：317
- 期：Complete
- 页码：109-121
- 全文大小：4010 K
文摘
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Juvenile sea bass brain is characterized by extensive active cell proliferation.
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Early life adversity (UCLIS) did not change juvenile brain cell proliferation.
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Acute stress reduced mitotic activity in teleost hippocampus, amygdala, cerebellum.
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Acute stress increased plasma cortisol in all cases (non-UCLIS, UCLIS juveniles).
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Early UCLIS modified acute stress plasticity of brain cell proliferation responses.