Astrocyte membrane properties are altered in a rat model of developmental cortical malformation but single-cell astrocytic glutamate uptake is robust
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- 作者：Elizabeth Hansona ; b ; Niels Christian Danboltc ; Chris G. Dullaa ; b ;
- 关键词：Epilepsy ; Cortical malformation ; Glutamate ; Glutamate transport ; GLT-1 ; GLAST ; EAAT1 ; EAAT2 ; Membrane resistance ; Glial development
- 刊名：Neurobiology of Disease
- 出版年：2016
- 出版时间：May 2016
- 年：2016
- 卷：89
- 期：Complete
- 页码：157-168
- 全文大小：1687 K
文摘
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Glutamate uptake by individual astrocytes is robust at all developmental stages in the freeze lesion model of developmental cortical malformation.
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Neonatal astrocyte membrane resistance is altered by freeze lesion.
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Regional and developmental distribution of glutamate transporters is altered by freeze lesion.
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Glutamatergic signaling is altered in adult freeze lesion cortex.
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