Disinhibition of neurons of the nucleus of solitary tract that project to the superior salivatory nucleus causes choroidal vasodilation: Implications for mechanisms underlying choroidal baroregulation
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- 作者：Chunyan Lia ; chli@uthsc.edu" class="auth_mail" title="E-mail the corresponding author ; Malinda E.C. Fitzgeralda ; b ; c ; malinda@cbu.edu" class="auth_mail" title="E-mail the corresponding author ; Nobel Del Mara ; ndelmar@uthsc.edu" class="auth_mail" title="E-mail the corresponding author ; Dr. Anton Reinera ; b ; areiner@uthsc.edu" class="auth_mail" title="E-mail the corresponding author
- 关键词：Ach ; acetylcholine ; ADN ; aortic depressor nerve ; ABP ; arterial blood pressure ; ChBF ; choroidal blood flow ; ChBVol ; choroidal blood volume ; ChBVel ; choroidal blood velocity ; CVLM ; caudal ventrolateral medulla ; Glu ; glutamate ; GAD ; glutamic acid decarboxylase ; NO ; nitric oxide ; NOS ; nitric oxide synthase ; NTS ; nucleus of solitary tract ; RVLM ; rostral ventrolateral medulla ; SSN ; superior salivatory nucleus ; VIP ; vasoactive intestinal polypeptide ; VGLUT2 ; vesicular glutamate transporter 2
- 刊名：Neuroscience Letters
- 出版年：2016
- 出版时间：28 October 2016
- 年：2016
- 卷：633
- 期：Complete
- 页码：106-111
- 全文大小：2114 K
文摘
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NTS neurons controlling choroidal blood flow receive dense inhibitory input.
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Disinhibition of NTS neurons causes increases in choroidal blood flow.
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Disinhibition of NTS during low blood pressure may drive choroidal vasodilation.
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Sign inversion of a low blood pressure signal may drive choroidal baroregulation.