Evidence for genistein as a mitochondriotropic molecule
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- 作者：Marcos Roberto de Oliveira mrobioq@gmail.com" class="auth_mail" title="E-mail the corresponding author mrobioq@yahoo.com.br" class="auth_mail" title="E-mail the corresponding author
- 关键词：8-OHdG ; 8-oxo-2&prime ; -deoxyguanosine ; AMPK ; AMP-activated protein kinase ; ANT ; adenine nucleotide translocator ; Bad ; Bcl2-associated agonist of cell death ; BKA ; bongkrekic acid ; CAT ; catalase ; CsA ; cyclosporine A ; Cu/Zn-SOD ; Cu/Zn-superoxide dismutase ; DR4 ; death receptor 4 ; DR5 ; death receptor 5 ; eNOS ; endothelial nitric oxide synthase ; ER ; endoplasmic reticulum ; ERα ; estrogen receptor α ; ERβ ; estrogen receptor β ; ERK1/2 ; extracellular signal-regulated kinase ; FADD ; FAS-associated death domain
- 刊名：Mitochondrion
- 出版年：2016
- 出版时间：July 2016
- 年：2016
- 卷：29
- 期：Complete
- 页码：35-44
- 全文大小：498 K
文摘
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Genistein interacts directly with mitochondrial proteins.
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Genistein exerts mitochondria-related antioxidant effects.
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Genistein induces mitochondrial biogenesis.
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Genistein triggers mitochondria-dependent cell death.
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Genistein may be a mitochondriotropic agent.
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