Chitosan Controls Postharvest Decay on Cherry Tomato Fruit Possibly via the Mitogen-Activated Protein Kinase Signaling Pathway

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• 作者：Danfeng Zhang ; Hongtao Wang ; Yi Hu ; Yongsheng Liu
• 刊名：Journal of Agricultural and Food Chemistry
• 出版年：2015
• 出版时间：August 26, 2015
• 年：2015
• 卷：63
• 期：33
• 页码：7399-7404
• 全文大小：389K
• ISSN：1520-5118

文摘

The inhibitive effects of chitosan on gray mold caused by Botrytis cinerea on cherry tomato fruit were evaluated. Decay incidence was tested on tomato stored at 22 掳C. Hydrogen peroxide accumulation, malondialdehyde (MDA) production, peroxidase (POD) activity, and several related gene expressions (including MPK3, MPK6, PR1a1, and PR5) were determined. Results showed that 0.2% of chitosan solution significantly inhibited the tomato gray mold 3 days after inoculation. Hydrogen peroxide accumulated in the fruit epidermal peel along with chitosan treatment, while MDA production was not increased. POD activity was remarkably enhanced by the application of chitosan. The relative expressions of MPK3, MPK6, and PR1a1 were significantly induced in 10 min after chitosan treatment, while PR5 was induced in 20 min. These findings suggested that the effects of chitosan on inhibiting gray mold in cherry tomato fruit were probably associated with the mitogen-activated protein kinase (MAPK) signaling pathway.