Addison’s disease with pituitary hyperplasia: a case report and review of the literature

详细信息    查看全文

• 作者：Jiaqiang Zhou (1)
  Lingxiang Ruan (2)
  Hong Li (1)
  Qingqing Wang (1)
  Fenping Zheng (1)
  Fang Wu (1)
  
• 关键词：Addison’s disease ; Hyperplasia ; Pituitary ; Replacement therapy
• 刊名：Endocrine
• 出版年：2009
• 出版时间：June 2009
• 年：2009
• 卷：35
• 期：3
• 页码：285-289
• 全文大小：269KB
• 参考文献：1. T. Kubota, M. Hayashi, M. Kabuto et al., Corticotroph cell hyperplasia in a patient with Addison disease: case report. Surg. Neurol. 37(6), 441-47 (1992) CrossRef
  2. K. Sugiyama, M. Kimura, T. Abe et al., Hyper-adrenocorticotropinemia in a patient with Addison’s disease after treatment with corticosteroids. Intern. Med. 35(7), 555-59 (1996) CrossRef
  3. K. Sekiya, H. Nawata, K. Kato et al., Diurnal rhythms of proopiomelanocortin-derived N-terminal peptide, beta-lipotropin, beta-endorphin and adrenocorticotropin in normal subjects and in patients with Addison’s disease and Cushing’s disease. Endocrinol. Jpn. 33(5), 713-19 (1986)
  4. G.A. Wittert, J.H. Livesey, C. Florkowski et al., Acutely raised corticotropin levels in Addison’s disease are not associated with increased plasma arginine vasopressin and corticotropin-releasing factor concentrations in peripheral plasma. J. Clin. Endocrinol. Metab. 76(1), 192-96 (1993) CrossRef
  5. K. Kovacs, L. Stefaneanu, E. Horvath et al., Pituitary corticotroph adenoma in a woman with long-standing Addison’s disease: a histologic, immunocytochemical, electron microscopic, and in situ hybridization study. Endocr. Pathol. 7(1), 91-7 (1996) CrossRef
  6. S. Miyabo, K. Miyanaga, K. Kimura et al., Inappropriate secretion of adrenocorticotropin from corticotroph hyperplasia in a case of Addison’s disease. Jpn. J. Med. 29(1), 38-5 (1990)
  7. K. Mineura, T. Goto, M. Yoneya et al., Pituitary enlargement associated with Addison’s disease. Clin. Radiol. 38(4), 435-37 (1987) CrossRef
  8. R. Clayton, A.C. Burden, V. Schrieber et al., Secondary pituitary hyperplasia in Addison’s disease. Lancet 2(8045), 954-56 (1977) CrossRef
  9. R.N. Dexter, D.N. Orth, K. Abe et al., Cushing’s disease without hypercortisolism. J. Clin. Endocrinol. Metab. 30(5), 573-79 (1970) CrossRef
  10. R.S. Gup, L.R. Sheeler, M.C. Maeder et al., Pituitary enlargement and primary hypothyroidism: a report of two cases with sharply contrasting outcomes. Neurosurgery 11(6), 792-94 (1982) CrossRef
  11. A.S. Joshi, P.D. Woolf, Pituitary hyperplasia secondary to primary hypothyroidism: a case report and review of the literature. Pituitary 8(2), 99-03 (2005) CrossRef
  12. B. Krautli, J. Muller, A.M. Landolt et al., ACTH-producing pituitary adenomas in Addison’s disease: two cases treated by transsphenoidal microsurgery. Acta. Endocrinol. 99(3), 357-63 (1982)
  13. W.W. Hutchins, J.V. Crues 3rd, P. Miya et al., MR demonstration of pituitary hyperplasia and regression after therapy for hypothyroidism. Am. J. Neuroradiol. 11(2), 410 (1990)
  14. A.J. Dwyer, J.A. Frank, J.L. Doppman et al., Pituitary adenomas in patients with Cushing disease: initial experience with Gd-DTPA-enhanced MR imaging. Radiology 163(2), 421-26 (1987)
  15. W.W. Peck, W.P. Dillon, D. Norman et al., High-resolution MR imaging of pituitary microadenomas at 15 T: experience with Cushing disease. Am. J. Roentgenol. 152(1), 145-51 (1989)
  16. J.D. Wilson, D.W. Foster, H.M. Kronenberg et al., / Williams textbook of endocrinology, 9th edn. (WB Sanders, Philadelphia, 1998), pp 547-63
  
• 作者单位：Jiaqiang Zhou (1)
  Lingxiang Ruan (2)
  Hong Li (1)
  Qingqing Wang (1)
  Fenping Zheng (1)
  Fang Wu (1)
  
  1. Department of Endocrinology, Sir Run Run Shaw Hospital, College of Medicine; Sir Run Run Shaw Institute of Clinical Medicine of Zhejiang University, Zhejiang University, 3 Qingchun East Road, Hangzhou, 310016, Zhejiang Province, People’s Republic of China
  2. Department of Radiology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou, 310003, People’s Republic of China
  

文摘

This case study describes a 33-year-old man with Addison’s disease who presented with increased plasma adrenocorticotropic hormone (ACTH), normal plasma cortisol, and absent diurnal rhythms. Magnetic resonance imaging (MRI) indicated pituitary hyperplasia. Conventional hydrocortisone replacement therapy may not inhibit high ACTH levels in the morning; however, replacing hydrocortisone with dexamethasone achieved good therapeutic results.