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Genetic diversity and phylogenetic analysis of glycoprotein gp85 of avian leukosis virus subgroup J wild-bird isolates from Northeast China
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  • 作者:Lili Jiang (1) (2) (3)
    Xiangwei Zeng (3)
    Yuping Hua (3)
    Qi Gao (1)
    Zhaobin Fan (2)
    Hongliang Chai (3)
    Qi Wang (1)
    Xiaole Qi (1)
    Yongqiang Wang (1)
    Honglei Gao (1)
    Yulong Gao (1)
    Xiaomei Wang (1)
  • 刊名:Archives of Virology
  • 出版年:2014
  • 出版时间:July 2014
  • 年:2014
  • 卷:159
  • 期:7
  • 页码:1821-1826
  • 全文大小:442 KB
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  • 作者单位:Lili Jiang (1) (2) (3)
    Xiangwei Zeng (3)
    Yuping Hua (3)
    Qi Gao (1)
    Zhaobin Fan (2)
    Hongliang Chai (3)
    Qi Wang (1)
    Xiaole Qi (1)
    Yongqiang Wang (1)
    Honglei Gao (1)
    Yulong Gao (1)
    Xiaomei Wang (1)

    1. State Key Laboratory of Veterinary Biotechnology, Division of Avian Infectious Diseases, Harbin Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Maduan Street 427, Harbin, 150001, China
    2. Institute of animal husbandry veterinary, Liaoning Medical University, Jinzhou, 121001, China
    3. College of Wildlife Resources, Northeast Forestry University, Harbin, 150040, China
  • ISSN:1432-8798
文摘
Avian leukosis virus subgroup J (ALV-J), first isolated in 1989, preferentially infects meat-type birds. Chinese layer flocks have experienced outbreaks of this virus since 2008. To analyze the status of ALV-J infection in wild birds in China, 585 wild birds collected from three provinces of Northeast China from 2010 to 2012 were tested, and six ALV-J strains were isolated for the first time. Furthermore, the gp85 genes of the six strains were amplified, cloned, and sequenced. The results indicated that two different ALV-J strains coexisted in Chinese wild birds from 2010 to 2012. These results not only expand the epidemiological data available for ALV-J and provide necessary information for the further understanding of the evolution of ALV-J, but they also highlight the potential role of wild-bird migration in the spread of ALV-J.

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