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Effect of interleukin‐1β and lipoxin A4 in human endometriotic stromal cells: Proteomic analysis
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文摘
Lipoxin A4 (LXA4) can function as an endogenous ‘breaking signal’ in inflammation and plays an important role in the progression of endometriosis. The proteome responses to interleukin-1β (IL-1β) or LXA4 in human endometriotic stromal cells (ESC) are not well understood.

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