Lateral parabrachial nucleus and opioid mechanisms of the central nucleus of the amygdala in the control of sodium intake
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- 作者：Glá ; ucia M.F. Andrade-Franzé ; Silvia Gasparini ; Laurival A. De Luca Jr. ; Patrí ; cia M. De Paula ; Dé ; bora S.A. Colombari ; Eduardo Colombari ; Carina A.F. Andrade ; José ; V. Menani ; menani@foar.unesp.br" class="auth_mail" title="E-mail the corresponding author ; carina.andrade@foar.unesp.br" class="auth_mail" title="E-mail the corresponding author
- 关键词：ANG II ; angiotensin II ; CAP ; captopril ; CeA ; central nucleus of amygdala ; FURO ; furosemide ; LPBN ; lateral parabrachial nucleus ; NTS ; nucleus of the solitary tract ; s.c. ; subcutaneously
- 刊名：Behavioural Brain Research
- 出版年：2017
- 出版时间：1 January 2017
- 年：2017
- 卷：316
- 期：Complete
- 页码：11-17
- 全文大小：1119 K
文摘
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CeA facilitatory and LPBN inhibitory mechanisms interact to control NaCl intake.
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Naloxone in the CeA blocks NaCl intake by euhydrated rats with muscimol into the LPBN.
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Naloxone in the CeA blocks NaCl intake by hyperosmotic rats with LPBN moxonidine.
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Opioids in the CeA are important for sodium intake induced by LPBN deactivation.
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Connection between LPBN and CeA mechanisms may depend on opioids action in the CeA.
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