Misexpression of Tbx18 in cardiac chambers of fetal mice interferes with chamber-specific developmental programs but does not induce a pacemaker-like gene signature
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- 作者：Franziska Greulicha ; Mark-Oliver Trowea ; Andreas Lefflerb ; Carsten Stoetzerb ; Henner F. Farina ; Andreas Kisperta ; kispert.andreas@mh-hannover.de" class="auth_mail" title="E-mail the corresponding author
- 关键词：ASD ; atrial septal defects ; AP ; action potential ; AVN ; atrioventricular node ; ChIP-Seq ; chromatin immunoprecipitation-sequencing ; Cx ; Connexin ; DAPI ; 4 ; 6-diamidino-2-phenylindol ; E ; embryonic day ; FC ; fold change ; Gapdh ; glyceraldehyde-3-phosphate dehydrogenase ; GFP ; green fluorescent protein ; Gjc ; gap junction protein ; gamma ; Gjd ; gap junction protein ; delta ; IRES ; internal ribosomal entry site ; Hcn ; hyperpolarization-activated cyclic nucleotide gated channel ; Hprt ; hypoxanthine guanine phospho
- 刊名：Journal of Molecular and Cellular Cardiology
- 出版年：2016
- 出版时间：August 2016
- 年：2016
- 卷：97
- 期：Complete
- 页码：140-149
- 全文大小：4142 K
文摘
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Misexpression of Tbx18 in chambers of fetal mice affects cardiac morphology and function.
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Misexpression of Tbx18 in chambers of fetal mice does not induce a pacemaker gene signature.
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Misexpression of Tbx18 in chambers of fetal mice affects the working myocardium gene program.
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